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Clinician-led medical education. Heart · Diabetes · Wellness.
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06/14/2026

Chronic stress = cardiovascular risk.

Sustained sympathetic activation drives hypertension, endothelial dysfunction, pro-inflammatory states, and arrhythmogenesis, and is associated with increased incidence of MI, arrhythmia, and Takotsubo cardiomyopathy.

In men, the clinical picture is shaped by higher sympathetic reactivity, lower rates of help-seeking, and frequent clustering with other modifiable risk factors. The result is a stress burden that often goes unaddressed until cardiovascular disease becomes clinically apparent.

Dr. Sahib Khalsa's talk on the sympathetic nervous system and stress is now available on https://hcp.medicalvisual.com/video-details/the-sympathetic-nervous-system-and-stress-bhns-, covering the underlying physiology, clinical implications, and emerging directions in autonomic-targeted care. Watch now!

06/11/2026

EP Live Austin 2026 is over, but the learning still continues!

If you missed the two days of live EP cases from St. David's Medical Center in Austin, get ready to watch at your own pace.

Sessions cover the latest ablation technology including emerging investigational platforms, trial results presented directly by the investigators, expert perspectives on EP, and live cases from the lab with real-time clinical decision-making.

Access the full EP Live conference videos only at hcp.medicalvisual.com. Coming soon!

06/05/2026

Secondary prevention of sudden cardiac death is built on a layered approach: addressing the substrate, suppressing the trigger, and protecting against recurrence.

ICDs remain the cornerstone for survivors of hemodynamically significant VT/VF without reversible cause.

Catheter ablation reduces arrhythmia burden and ICD therapies in scar-related VT, with growing use earlier in the disease course.

Coronary angiography and revascularization when indicated addresses ischemic triggers, though it does not replace device therapy in most survivors.

Pharmacotherapy with beta-blockers, amiodarone, mexiletine, or sotalol complements these interventions based on substrate and tolerability.

The goal is not a single definitive treatment, but a coordinated strategy matched to mechanism.

Learn more at hcp.medicalvisual.com!

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